@article{Chereshnyuk_2017, title={Efficiency ademol in reperfusion period of total ischemia eye through treatment principles of modern neyroretynoprotective therapy}, volume={19}, url={https://reports-vnmedical.com.ua/index.php/journal/article/view/154}, abstractNote={<p>In this paper is presented the synthesis of recent data about pathobiochemical features neurons damaged retina and optic nerve in terms of their ischemic-hypoxic damage of various genesis and modern possibilities of pharmacological action on key target receptor on the example Ademol - neuroprotective agent with a modulating effect on the functioning of NMDA-receptors’ activity and selective blockade of β-adrenergic receptors. In terms of the model 60 minutes total ischemia-reperfusion of eye installed capacity of Ademol (2 mg/kg) reduced the level of activity of membrane integrity marker damage neurons of the retina - the neuron-specific enolase, indicating the presence of a study drug neuroretinoprotective properties.</p&gt;}, number={1}, journal={Reports of Vinnytsia National Medical University}, author={Chereshnyuk, I.L.}, year={2017}, month={Sep.}, pages={14-20} }